FGSC #949

Mutant Type

Genus: N

reporting_genes: Has been replaced by strain number: 1169

species: Neurospora crassa

allele: 44411;Y30539y;no#

stock: 9065

glasgow:

mutagen:

Depositor: DDP

Link Group: VR;VIL;VII

MT: A

Species No: 10

gene_back:

oppmt: 0

trans:

ref1:

ref2:

site:

country:

ksudc_link: https://digital.lib.k-state.edu/item/neurospora-crassa/fgsc-949

Locus	Cultural Requirements	Link Group	Type
ylo-1	VIL. Between cys-1 (8%) and ad-1 (6%). Probably right of Bml (2%) (1012, PB). (381). Yellow carotenoids (381). Affects synthesis of neurosporaxanthin (4'-apo-beta'-caroten-4'-oic acid); citations in reference 398. Lesion probably involves the conversion of lycopene to 3,4-dehydrolycopene or the conversion of either torulene or gamma-carotene to neurosporaxanthin (398 and references therein) (Fig. 9). Resembles the orange wild type in young cultures, but color differences become clear with age. Expressed in both conidia and mycelia. Undefined modifiers affect intensity. Fails to complement with many of the al-1 and al-2 albino strains (R.E. Subden, personal communication).	VIL	B
ad-7	VR. Right of cot-2 (4%). Left of ro-4 (4%) and pab-2(8%). (158, 156). (687) Uses adenine or hypoxanthine (682). Lacks phosphoribosylpyrophosphate amidotransferase, the first enzyme in de novo purine biosynthesis (525) (Fig. 8). Ascospores from homozygous ad-7 x ad-7 crosses are white (allele Y175M256). Strains carrying allele P73Bl7l(t) are temperature sensitive.	VR	B
slo-2	VII. Left of met-7 (2%) (816). (W.N. Strickland, cited in reference 8I6) Slow growth from conidia or ascospores. Conidiation lags days behind that of the wild type (816, 1035). Not tested for cytochrome deficiency or ability to reduce tetrazolium.	VII	B

Neurospora Crassa Wikipedia