FGSC #3149

Mutant Type

Genus: N

reporting_genes: T(I->II;I;VII)AR217,ad-9;cyh-1

species: Neurospora crassa

allele: AR217 Y154M37 KH52(r)

stock: 127-1230

glasgow:

mutagen:

Depositor: DDP

Link Group: IR;VII;II;IR IR

MT: a

Species No: 10

gene_back:

oppmt: 0

trans:

ref1:

ref2:

site:

country:

ksudc_link: https://digital.lib.k-state.edu/item/neurospora-crassa/fgsc-3149

Locus	Cultural Requirements	Link Group	Type
ad-9	IR. Right of met-6 (9 to 16%). Left of Tp(T54M94) and nit-1 (3 to 15%) (466, 816). (815) Uses adenine or hypoxanthine (526). Controls conversion of phosphoribosylglycineamide to phosphoribosylformylglycineamide (120) (Fig. 8). Called complementation group D.	IR	B
cyh-1	IR. Right of nit-1 (6%). Left of T(STL76) and al-2 (8 to 13%) (496, 797, 808).Resistant to cycloheximide (496, 748). Resistance is recessive in duplications (1090). Dominance reported in forced heterokaryons (496, 748) may have been due to skewed nuclear ratios (1090). Protein synthesis on ribosomes of the mutant cyh-1 proceeds in the presence of cycloheximide in a cell-free system (834). Readily scored on slants with 10 µg of cycloheximide per ml autoclaved in the medium. Excellent as a marker and valuable for selecting somatic recombinants or deletions in heterozygous duplications (748, 1091). Used to show that the cycloheximide-induced phase shift of the circadian clock involves protein synthesis (738). Called act-1: actidione resistant-1.	IR	B
T(I->II;I;VII)AR217	Translocation		B

Neurospora Crassa Wikipedia