FGSC #3418

Mutant Type

Genus: N

reporting_genes: T(V^VII)17-088 Pk

species: Neurospora crassa

allele: 17-088

stock: 17-088-11

glasgow:

mutagen:

Depositor: AMS

Link Group: V;VII

MT: a

Species No: 10

gene_back:

oppmt: 0

trans:

ref1: Russell & Srb 1972 Genetics 71:233-245, https://doi.org/10.1093/genetics/71.2.233

ref2: Srb et al 1973 J Hered 64:242-246, https://doi.org/10.1093/oxfordjournals.jhered.a108404

site:

country:

ksudc_link: https://digital.lib.k-state.edu/item/neurospora-crassa/fgsc-3418

ksudc_link_html: https://digital.lib.k-state.edu/item/neurospora-crassa/fgsc-3418 ↗

Genes

Locus	Cultural Requirements	Link Group	Type
T(V^VII)17-088	Translocation		B
pk	bis, biscuit) VR. Between met-3 (1%) and T(EB4)^L, cot-2 (8%), cl (2%). Growth on an agar surface is initially colonial and flat. A mass of aerial hyphae is then sent up, which conidiates profusely (1548). Somewhat similar in morphology to sn, cum, sp, and cot-4 at 25ºC, but distinguishable. Hyphae branch dichotomously (1405, 1548). Increased activity of L-glutamine:D-fructose-6-phosphate amidotransferase was observed in crude extracts of one pk strain, but not in nine others; increased activity for this enzyme also was found in cl and in four other nonallelic morphological mutants (1758). Hexoseaminoglycan consists of a single component on medium without sorbose, in contrast to two components in wild type (1960). Antigenic surface mucopolyoside (532). Cell-wall analysis and photograph, allele B6 (507) and allele C-1810-1 (287). Cell-wall enzymes (633). Effect of carbon source (531). One observation suggested a functional interaction with cl (1965), but substantial crossing-over frequencies and recovery of the double mutant pk cl indicated that the loci are distinct (569). The gene was named for the vegetative mutant phenotype, which is recessive. Several alleles were called bis (1592). The sexual phase is also affected. Asci are thin-walled, bulbous, and nonlinear in homozygous pk ´ pk crosses (1406, 1409, 1550). Spindle orientation is abnormal in the swollen asci, and no apical pore is formed (1625, 1679). Most mutant alleles are recessive for the ascus effect, but some are dominant with variable penetrance. Sorbose-resistant mutants at various loci act as dominance modifiers of the ascus effect of dominant alleles (1757). Sexual-phase-recessive allele C-1610 and dominant allele 17-088 are both associated with reciprocal translocations (1578).		B