FGSC #7781

Mutant Type

Genus: N

reporting_genes: This strain is not available ace-8 met-7

species: Neurospora crassa

allele: KG570 4894

stock: m570a

glasgow:

mutagen:

Depositor: HK

Link Group: VIIL VIIR

MT: a

Species No: 10

gene_back: SL

oppmt: 0

trans:

ref1: Kuwana and Tanaka 1987 Jpn J Genet 62:283-289, https://doi.org/10.1266/jjg.62.283

ref2: Kuwana and Kubota 1983 Jpn J Genet 58:579-589, https://doi.org/10.1266/jjg.58.579

site:

country:

ksudc_link: https://digital.lib.k-state.edu/item/neurospora-crassa/fgsc-7781

Locus	Cultural Requirements	Link Group	Type
met-7	VIIR. Right of qa-2 (<1%), ars (<1%), and the centromere (one second-division ascus in several hundred). Left of met-9 (10[-4]) and wc-1 (1 to 4%) (146, 725; M.E. Case, personal communication). (718; M.K. Allen, cited in references 718 and 789) Uses cystathionine, homocysteine, or methionine (718; N. H. Horowitz, cited in reference 1180). Lacks cystathionine-gamma-synthase (547) (Fig. 17). This enzyme is also lacking in the mutant met-3 (547). See met-3 for regulation. Apparently contiguous with met-9by coconversion. Flanking markers are recombined in most met-7+ met-9+ recombinants (725). Functionally distinct from the mutant met-9, which has active cystathionine-gamma-synthase (547) but cannot use homocysteine. No mutants lacking both functions have been isolated. Allele NM251 is suppressible by supersuppressor RN33 (same as ssu-1?) (725). Allele K79 is inseparable from reciprocal translocation T(I;VII)K79 (808).	VIIR	B
ace-8	Requires acetate. Inhibited by sucrose.	VIIL	B

Neurospora Crassa Wikipedia